Search results for "Brief Definitive Report"

showing 10 items of 14 documents

Cytomegalovirus Misleads Its Host by Priming of CD8 T Cells Specific for an Epitope Not Presented in Infected Tissues

2003

Cytomegaloviruses (CMVs) code for several proteins that inhibit the presentation of antigenic peptides to CD8 T cells. Although the molecular mechanisms of CMV interference with the major histocompatibility complex class I pathway are long understood, surprisingly little evidence exists to support a role in vivo. Here we document the first example of the presentation of an antigenic peptide being blocked by a CMV immune evasion protein in organs relevant to CMV disease. Although this Db-restricted peptide, which is derived from the antiapoptotic protein M45 of murine CMV (mCMV), is classified as an immunodominant peptide based on response magnitude and long-term memory, adoptive transfer of…

Adoptive cell transferImmunologyMutantCytomegalovirusPriming (immunology)PeptideCD8-Positive T-LymphocytesBiologyLymphocyte ActivationMajor histocompatibility complexEpitopeImmune systemHumansImmunology and AllergyCytotoxic T cellimmune evasionchemistry.chemical_classificationimmune controlimmunodominanceImmunomagnetic SeparationBrief Definitive Reportvirus diseasesAdoptive TransferVirologyantigen presentationchemistryCytomegalovirus InfectionsImmunologybiology.proteincross-primingImmunologic MemoryJournal of Experimental Medicine
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NFATc2 and NFATc3 transcription factors play a crucial role in suppression of CD4+ T lymphocytes by CD4+ CD25+ regulatory T cells

2005

The phenotype of NFATc2(-/-) c3(-/-) (double knockout [DKO]) mice implies a disturbed regulation of T cell responses, evidenced by massive lymphadenopathy, splenomegaly, and autoaggressive phenomena. The population of CD4(+) CD25(+) T cells from DKO mice lacks regulatory capacity, except a small subpopulation that highly expresses glucocorticoid-induced tumor necrosis factor receptor family-related gene (GITR) and CD25. However, neither wild-type nor DKO CD4(+) CD25(+) regulatory T cells (T reg cells) are able to suppress proliferation of DKO CD4(+) CD25(-) T helper cells. Therefore, combined NFATc2/c3 deficiency is compatible with the development of CD4(+) CD25(+) T reg cells but renders c…

CD4-Positive T-LymphocytesT cellImmunologyPopulationchemical and pharmacologic phenomenaReceptors Nerve Growth FactorBiologyLymphocyte ActivationReceptors Tumor Necrosis FactorInterleukin 21MiceT-Lymphocyte SubsetsGlucocorticoid-Induced TNFR-Related ProteinmedicineImmunology and AllergyCytotoxic T cellAnimalsIL-2 receptorReceptoreducationTranscription factorMice Knockouteducation.field_of_studyNFATC Transcription FactorsZAP70Brief Definitive ReportNuclear Proteinshemic and immune systemsReceptors Interleukin-2Molecular biologyCoculture TechniquesDNA-Binding Proteinsmedicine.anatomical_structureTranscription FactorsThe Journal of Experimental Medicine
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Identification of modifications in microbial, native tRNA that suppress immunostimulatory activity

2012

2′-O-methylation of guanosine 18 is a naturally occurring tRNA modification that can suppress immune TLR7 responses.

ImmunologyMutantfungiBrief Definitive ReportRNAfood and beveragesvirus diseasesContext (language use)Biologybiochemical phenomena metabolism and nutritionmedicine.disease_causeTRNA MethyltransferasesTransplantationchemistry.chemical_compoundBiochemistrychemistryTransfer RNAmedicineImmunology and AllergyEscherichia coliDNAThe Journal of Experimental Medicine
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Cyclic adenosine monophosphate is a key component of regulatory T cell–mediated suppression

2007

Naturally occurring regulatory T cells (T reg cells) are a thymus-derived subset of T cells, which are crucial for the maintenance of peripheral tolerance by controlling potentially autoreactive T cells. However, the underlying molecular mechanisms of this strictly cell contact–dependent process are still elusive. Here we show that naturally occurring T reg cells harbor high levels of cyclic adenosine monophosphate (cAMP). This second messenger is known to be a potent inhibitor of proliferation and interleukin 2 synthesis in T cells. Upon coactivation with naturally occurring T reg cells the cAMP content of responder T cells is also strongly increased. Furthermore, we demonstrate that natur…

Interleukin 2CD4-Positive T-LymphocytesMaleRegulatory T cellImmunologyEnzyme-Linked Immunosorbent AssayBiologySecond Messenger SystemsT-Lymphocytes RegulatoryConnexinschemistry.chemical_compoundMiceImmune systemmedicineCyclic AMPSuppressor Factors ImmunologicImmunology and AllergyAnimalsCyclic adenosine monophosphateIL-2 receptorDNA PrimersMice Inbred BALB CReverse Transcriptase Polymerase Chain ReactionZAP70Intercellular transportBrief Definitive ReportPeripheral toleranceGap JunctionsMolecular biologyMice Inbred C57BLmedicine.anatomical_structurechemistryBrief Definitive ReportsCytokinesFemaleOligopeptidesmedicine.drugThe Journal of Experimental Medicine
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Multicolor fate mapping of Langerhans cell homeostasis

2013

The adult epidermal Langerhans cell network is formed by adjacent proliferative units composed of dividing cells and their terminally differentiated daughter cells.

Langerhans cellCell divisionImmunologyPopulationCellCytological TechniquesColorMice TransgenicBiology03 medical and health sciencesMice0302 clinical medicineImmune systemImaging Three-DimensionalFate mappingmedicineImmunology and AllergyAnimalsHomeostasisCell Lineageeducation030304 developmental biologyInflammation0303 health scienceseducation.field_of_studyEpidermis (botany)integumentary systemBrief Definitive ReportCell biologyMice Inbred C57BLmedicine.anatomical_structure030220 oncology & carcinogenesisLangerhans CellsImmunologyStem cellJournal of Experimental Medicine
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Langerhans cells are negative regulators of the anti-Leishmania response

2011

Langerhans cells suppress the immune response to low-dose Leishmania major infection in part by inducing regulatory T cells.

LangerinT cellImmunologyPriming (immunology)Leishmaniasis Cutaneouschemical and pharmacologic phenomenaT-Lymphocytes RegulatoryImmune toleranceInterferon-gammaMiceImmune systemSDG 3 - Good Health and Well-beingparasitic diseasesmedicineImmune ToleranceImmunology and AllergyAnimalsInterferon gammaLeishmania majorLeishmaniasis VaccinesLeishmania majorbiologyintegumentary systemBrief Definitive ReportFOXP3hemic and immune systemsbiochemical phenomena metabolism and nutritionTh1 Cellsbiology.organism_classificationDisease Models Animalmedicine.anatomical_structureLangerhans CellsImmunologybiology.proteinmedicine.drug
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Resistance of natural killer T cell-deficient mice to systemic Shwartzman reaction.

2000

The generalized Shwartzman reaction in mice which had been primed and challenged with lipopolysaccharide (LPS) depends on interleukin (IL)-12-induced interferon (IFN)-gamma production at the priming stage. We examined the involvement in the priming mechanism of the unique population of Valpha14 natural killer T (NKT) cells because they promptly produce IFN-gamma after IL-12 stimulation. We report here that LPS- or IL-12-primed NKT cell genetically deficient mice were found to be resistant to LPS-elicited mortality. This outcome can be attributed to the reduction of IFN-gamma production, because injection of recombinant mouse IFN-gamma, but not injection of IL-12, effectively primed the NKT …

LipopolysaccharidesShwartzman phenomenonReceptors Antigen T-Cell alpha-betaImmunologyPopulationPriming (immunology)Mice SCIDBiologyLymphocyte DepletionInterferon-gammaMiceInterferonmedicineImmunology and AllergyAnimalsInterferon gammaLectins C-TypeAntigenseducationeducation.field_of_studyMice Inbred BALB Cinterferon γTumor Necrosis Factor-alphalipopolysaccharideBrief Definitive ReportInterleukinProteinsShwartzman reactionNatural killer T cellmedicine.diseaseInterleukin-12Immunity Innatenatural killer T cellsKiller Cells NaturalMice Inbred C57BLImmunologyAntigens SurfaceInterleukin 12interleukin 12medicine.drugNK Cell Lectin-Like Receptor Subfamily BShwartzman PhenomenonThe Journal of experimental medicine
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The nuclear receptor PPARγ selectively inhibits Th17 differentiation in a T cell–intrinsic fashion and suppresses CNS autoimmunity

2009

T helper cells secreting interleukin (IL)-17 (Th17 cells) play a crucial role in autoimmune diseases like multiple sclerosis (MS). Th17 differentiation, which is induced by a combination of transforming growth factor (TGF)-beta/IL-6 or IL-21, requires expression of the transcription factor retinoic acid receptor-related orphan receptor gamma t (ROR gamma t). We identify the nuclear receptor peroxisome proliferator-activated receptor gamma (PPAR gamma) as a key negative regulator of human and mouse Th17 differentiation. PPAR gamma activation in CD4(+) T cells selectively suppressed Th17 differentiation, but not differentiation into Th1, Th2, or regulatory T cells. Control of Th17 differentia…

MESH: Nuclear Receptor Subfamily 1 Group F Member 3Helper-InducerReceptors Retinoic AcidT-LymphocytesMESH: Interleukin-17Cellular differentiationRetinoic AcidPeroxisome proliferator-activated receptorNeurodegenerativeInbred C57BLMedical and Health SciencesMiceInterleukin 210302 clinical medicineGroup FRAR-related orphan receptor gammaMESH: Nuclear Receptor Co-Repressor 2Receptors2.1 Biological and endogenous factorsThyroid HormoneImmunology and AllergyMESH: AnimalsAetiologyEncephalomyelitisPromoter Regions Geneticchemistry.chemical_classificationOrphan receptor0303 health sciencesReceptors Thyroid HormoneInterleukin-17Cell DifferentiationT-Lymphocytes Helper-InducerNuclear Receptor Subfamily 1 Group F Member 33. Good healthCell biologyDNA-Binding Proteinsmedicine.anatomical_structureMESH: Repressor Proteins[SDV.IMM]Life Sciences [q-bio]/ImmunologyInterleukin 17MESH: Cell Differentiationmedicine.medical_specialtyEncephalomyelitis Autoimmune ExperimentalMultiple SclerosisNuclear Receptor Subfamily 1Member 31.1 Normal biological development and functioningT cellImmunologyBiologyAutoimmune DiseasePromoter RegionsExperimental03 medical and health sciencesGeneticUnderpinning researchMESH: Mice Inbred C57BLInternal medicineMESH: Promoter Regions GeneticGeneticsmedicineAnimalsHumansNuclear Receptor Co-Repressor 2MESH: Receptors Thyroid HormoneMESH: T-Lymphocytes Helper-InducerMESH: Encephalomyelitis Autoimmune ExperimentalMESH: Mice030304 developmental biologyMESH: Receptors Retinoic AcidMESH: HumansInflammatory and immune systemNeurosciencesBrief Definitive ReportCorrectionMESH: Multiple SclerosisBrain DisordersMice Inbred C57BLPPAR gammaRepressor ProteinsEndocrinologyMESH: PPAR gammaNuclear receptorchemistryMESH: DNA-Binding Proteins030217 neurology & neurosurgeryAutoimmuneJournal of Experimental Medicine
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Comprehensive three‐dimensional morphology of neoangiogenesis in pulmonary veno‐occlusive disease and pulmonary capillary hemangiomatosis

2019

Abstract Pulmonary veno‐occlusive disease (PVOD) is a rare lung disease characterized by fibrotic narrowing of pulmonary veins leading to pulmonary hypertension (PH) and finally to death by right heart failure. PVOD is often accompanied by pulmonary capillary hemangiomatosis (PCH), a marked abnormal proliferation of pulmonary capillaries. Both morphological patterns often occur together and are thought to be distinct manifestations of the same disease process and accordingly are classified together in group 1′ of the Nice classification of PH. The underlying mechanisms of these aberrant remodeling processes remain poorly understood. In this study, we investigated the three‐dimensional struc…

Pathologymedicine.medical_specialtyLung NeoplasmsHypertension Pulmonarypulmonary veno‐occlusive diseasePulmonary capillary hemangiomatosis030204 cardiovascular system & hematologypulmonary capillary hemangiomatosisPathology and Forensic Medicine03 medical and health sciencesThree dimensional morphology0302 clinical medicineRight heart failurepulmonary hypertensionmedicinelcsh:PathologyHumansHemangioma CapillaryLungNeovascularization Pathologicbusiness.industryBrief Definitive Reportintussusceptive neoangiogenesismedicine.diseasePulmonary hypertension3. Good healthmedicine.anatomical_structurePulmonary VeinsLung disease030220 oncology & carcinogenesisPulmonary Veno-Occlusive DiseaseImmunohistochemistryPulmonary Veno-Occlusive Diseasebusinesspulmonary vascular remodelinglcsh:RB1-214The Journal of Pathology: Clinical Research
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Protection from lethal septic peritonitis by neutralizing the biological function of interleukin 27

2006

The immune response to bacterial infections must be tightly controlled to guarantee pathogen elimination while preventing tissue damage by uncontrolled inflammation. Here, we demonstrate a key role of interleukin (IL)-27 in regulating this critical balance. IL-27 was rapidly induced during murine experimental peritonitis induced by cecal ligation and puncture (CLP). Furthermore, mice deficient for the EBI3 subunit of IL-27 were resistant to CLP-induced septic peritonitis as compared with wild-type controls, and this effect could be suppressed by injection of recombinant single-chain IL-27. EBI3−/− mice displayed significantly enhanced neutrophil migration and oxidative burst capacity during…

Recombinant Fusion ProteinsImmunologyDown-RegulationPeritonitisInflammationPeritonitisBiologySepsisMiceImmune systemSepsismedicineAnimalsImmunology and AllergyInterleukin 27Innate immune systemBacteriaInterleukinsBrief Definitive ReportInterleukinReceptors Interleukinmedicine.diseaseImmunity InnateUp-RegulationRespiratory burstMice Inbred C57BLProtein SubunitsSolubilityImmunologyBrief Definitive Reportsmedicine.symptomGranulocytesJournal of Experimental Medicine
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